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Nazzy Pakpour Faculty Profile

Photo of Nazzy Pakpour

Nazzy  Pakpour

Assistant Professor

Department of Biological Sciences

  • E-mail: nazzy.pakpour@csueastbay.edu
  • Phone: 510-885-2629
  • Office: SC-N 418
  • Office Hours: Wednesdays 2:00-4:00 pm or by appointment
  • Home Page: http://www.pakpourlab.com/
    Note: CSUEB does not pre-approve, monitor, or edit personal pages. Faculty members are solely responsible for their content, and are expected to conform to the policy guidelines of CSUEB.

My laboratory is currently working on the following projects:

1. Examining the impact of type 2 diabetes on malaria parasite development and transmission

2. Using hackathons as an educational and community building activity for college students

3. Establishing an insect petting zoo to travel to local schools 

For more information about any of these projects, my lab, or the classes I teach please visit my website.

  • Ph.D. University of Pennsylvania (2008)
  • B.S. University of California Davis (1999)
Spring Semester 2020
Course #SecCourse TitleDaysFromToLocationCampus
BIOL 43101Medical MicrobiologyWF9:30AM10:45AMSC-N108Hayward Campus
BIOL 4311AMedical MicrobiologyWF11:00AM1:50PMSC-S307Hayward Campus
BIOL 4311BMedical MicrobiologyWF3:00PM5:50PMSC-S307Hayward Campus
BIOL 49005Independent StudyARRARRHayward Campus

The following are a list of my most recent publications, a full list can be found on my website.

Tandon J, Gumina M, Pakpour N, Akhavian R. (2017) CSU East Bay Hack Day: a university hackathon to combat malaria and zika with drones. IEEE Global Engineering Education Conference (EDUCON). 

Pietri JE, Pakpour N, Napoli E, Song G, Pietri E, Potts R, Cheung KW, Walker G, Riehle MA, Starcevich H, Giulivi C, Lewis EE, Luckhart S. (2016) Two insulin-like peptides differentially regulate malaria parasite infection in the mosquito through effects on intermediary metabolism. Biochem J. 473.20: 3487-3503.

Luckhart S, Pakpour N, Giulivi C. (2015) Host-pathogen interactions in malaria: cross-kingdom signaling and mitochondrial regulation. Curr Opin Immunol. 36:73-9.

Wang B, Pakpour N, Napoli E, Drexler A, Glennon EK, Surachetpong W, Cheung K, Aguirre A, Klyver JM, Lewis EE, Eigenheer R, Phinney BS, Giulivi C, Luckhart S. (2015) Anopheles stephensi p38 MAPK signaling regulates innate immunity and bioenergetics during Plasmodium falciparum infection. Parasit Vectors, 8(1).

Neafsey DE et al. [including Pakpour N]. (2015). Highly evolvable malaria vectors: the genomes of 16 Anopheles mosquitoes. Science, 347(6217) :1258522.

Pakpour N, Luckhart S, Riehle MA. (2014) Effects of vertebrate-derived blood factors on insect immune responses. Curr Opin Ins Sci. 3:1-5.

Jiang X et al. [including Pakpour N]. (2014). Genome analysis of a major urban malaria vector mosquito, Anopheles stephensi. Genome Biol, 15:459.

Drexler AL, Pietri JE, Pakpour N, Hauck E, Wang B, et al. (2014) Human IGF1 regulates midgut oxidative stress and epithelial homeostasis to balance lifespan and Plasmodium falciparum resistance in Anopheles stephensi. PLoS Pathog, e1004231.

Pakpour N, Camp L, Smithers HM, Wang B, Tu Z, Adler SA, Luckhart SL. (2013) Protein kinase C-dependent signaling controls the midgut epithelial barrier to malaria parasite infection in anopheline mosquitoes. PLoS One 8: e76535.

Vodovotz Y, Azhar N, Miskov-Zivanov N, Buliga M, Zamora R, Ermentrout B, Constantine G, Faeder J, Pakpour N, Luckhart S. (2013) Modeling host-vector-pathogen immuno-inflammatory interactions in malaria., in: G. An and Y. Vodovotz (Ed.), Complex Systems and Computational Biology Approaches to Acute Inflammation, Springer Science & Business Media, New York, NY.

Chau JY, Lawrence JA, Tiffany CM, Mooney JP, Lokken KL, Pakpour N, Tsolis RM, Luckhart S. (2013) Malaria-associated L-Arginine deficiency induces mucosal mast cell-dependent disruption to the intestinal barrier defenses against non-typhoidal Salmonella bacteremia. Infect Immun. 81(10):3515-26.

Hauck E, Antonova-Koch Y, Drexler A, Pietri J, Pakpour N, Liu D, Blacutt J, Riehle MA, Luckhart S. (2013) Overexpression of phosphatase and tensin homolog (PTEN) improves fitness and decreases Plasmodium falciparum development in Anopheles stephensi. Microbes Infect. 15(12):775–787.

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